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At week 13, significant, dose-dependent reductions in HbA 1c (the primary endpoint) were reported [10]

Endothelial dysfunction has been demonstrated in vivo (306, 311), in the large vessels of ME/CFS patients (307, 311) through flow-mediated dilation (FMD) and in the microcirculation through post-occlusive reactive hyperaemia

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Laboratory research demonstrates interactions with specific molecular pathways in experimental models

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